A capillary hemangioma usually occurs in a nodular shape, and it is not common to have multiple lesions. A 57-year-old man presented with multiple disseminated erythematous papules and plaques on the trunk, which developed two months ago. Ten months ago, he was diagnosed with bcr-abl positive chronic myeloid leukemia(CML), and began taking bcr-abl tyrosine kinase inhibitors. He also had tuberculous lymphadenopathy and diabetes mellitus. On physical examination, erythematous rod shaped plaques developed mainly on the trunk. Skin biopsy was done on the chest. The biopsy revealed capillary proliferation in upper dermis, which is consistent with capillary hemangioma. Five months later when he returned to the clinic, the initial lesions did not disappear, and new lesions appeared even though he stopped taking the drug for CML. Since there is no temporal relationship between the drugs and hemangioma, and no known mechanism to cause the lesion, the possibility that the lesion may have occurred in relation to the angiogenic effect of the bcr-abl positive cell was suggested. According to previous studies, bcr-abl positive CML cells secreted more angiogenic factors than normal cells, and an increase in the angiogenic factors in patients suffering from both tuberculous and diabetes mellitus have been observed. Therefore, this case shed light on the possibility that the cause of hemagioma could be found in the comorbidities with angiogenic effect.