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기능성 소화불량의 증상 발현인자로서 스트레스의 의미
Role of Stress in Functional Dyspepsia기능성 소화불량의 증상 발현인자로서 스트레스의 의미
김정욱 ( Jung Wook Kim ) , 김시영 ( Si Young Kim ) , 김창섭 ( Chang Sup Kim ) , 이준행 ( Jun Haeng Lee ) , 조용균 ( Yong Kyun Cho ) , 박창영 ( Chang Young Park ) , 손정일 ( Chong Il Sohn ) , 전우규 ( Woo Kyu Jeon ) , 김병익 ( Byung Ik Kim ) , 정을순 ( Eul Sun Jung ) , 백상빈 ( Sang Bin P
UCI I410-ECN-0102-2009-510-003981947

Pathogenesis of functional dyspepsia consists of motor abnormality, sensory abnormality, and psychologic abnormality. Anxiety, depression, and psychosomatic disorders are well known psychopathologies that lead to dyspepsia, and stress seems to be an additional etiology. The aim of this study is to assess whether stress can cause and/or provoke dyspepsia. Methods: Functional dyspepsia was defined and classified by Rome criteria. SCL-90-R for the detection of psychopathology, measurement of life stress scale by self-administered questionnaire, and measurement of stress coping mechanisms using a multidimensional stress coping scale test were performed in 79 functional dyspeptic patients and 25 normal controls. Results: No significant difference was found in the total amount of stress between functional dyspeptic patients and the normal control. However, the patients were more stressful about issues relating to health and marital relationship among stresses in individual daily life. As the patients become more stressful, they used more negative copingmechanisms such as restraint, fatalism, criticism, religiousness, and negativism. These seem to provoke several psychopathologies including interpersonal sensitivity, depression, anxiety, hostility. However, there was no relationship in the amount of stress, the stress coping mechanism, and psychopathology among the dyspeptic symptoms and subgroups of functional dyspepsia. Conclusion: There was no difference in the total amount of stress between functional dyspeptic patients and the normal control. In some individuals who utilize inadequate coping mechanisms against stress, the stress can provoke dyspepsia. (Kor ean J our nal of Gastr ointestinal Motility 2002;8:146-152)

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