Traumatic asphyxia is a clinical symptom complex characterized hy craniofacial cyanosis subconjunctival hemorrhage, and head and neck vascular engorgement due to sudden compressive injury on the thoracic cage. It is occationally comhined with mental deterioration. Lune contusion, and edema. It is considered due to increa.ed intrathoracic pressure in state of' closed epiglottis. Recently, we had experienced three cases of traumatic asphyxia of which clinical courses were .omewhat differenl. The first case developed hy a compression between a elevator and the ground while the victim did not realized the accident happening,and the patient showed nonspccihc facial edem,I;ind ecchymosis but mental deterioration and ultimate respiratory failure. The second case by a compresion between cars, while the impending accident was noticed by the victim. Showed full-blown asphyxia without mentul or respiratory symptoms. The last case by forceful coughing and vomiting showed facial edema and ecchymosis without any other symptoms. Hospital courses of all cases were uneventful. We believe that "fear response" or closure of the epielottis" might be an important mechanism on developing symptom of traumatic asphyxia.